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J Am Chem Soc 133:1155711571

We next investigated the effect of BPC-157 on the HUVEC cell cycle distribution

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Dixon's group demonstrated that the GSH exporter MRP1 sensitizes HPA1 erastin-treated cells to ferroptosis and concordantly, MRP1 inhibition leads to a retention of GSH and leads to ferroptosis resistance (58)

Interestingly, activation/inactivation of glucocorticoids and 11-oxygenated androgens work in an antiparallel manner, with the 11-hydroxy derivative being the active glucocorticoid [74,75], while the 11-keto androgens are more potent than their 11-hydroxy counterparts [76]