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Existing research suggests that this could be another potential point of regulation for ferroptosis, by modulating the enzymes involved in the biosynthesis of membrane phospholipids containing PUFAs to trigger or block ferroptosis (39, 40)

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PMCID: PMC8176521

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Por tanto, la suplementacion con GHK-Cu exogeno tiene como objetivo restaurar las vias de senalizacion juveniles que gobiernan la reparacion y la defensa antioxidante