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Prior to the trial, the older cohort exhibited severe intracellular glutathione deficiency, massive oxidative stress, and impaired mitochondrial function compared to young, healthy controls

We therefore suggest a mechanism that can be described according to the Cleland nomenclature as a combination of a random bi-uni mechanism and a ping-pong bi-bi (or uni-uni-uni-uni) mechanism in which the single product of the rate-limiting random mechanism becomes the first substrate of the ping-pong mechanism (Fig

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