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Receptor Sequestration in Response to -Arrestin-2 Phosphorylation by ERK1/2 Governs Steady-State Levels of GPCR Cell-Surface Expression

To further develop our understanding of the impact of plasma treatment on GSH and GSSG as the central cellular redox system, we utilized the iron(II) complex ferrocenecarboxylic acid (complex 1 ) and the iron(III) complexes iron(III) acetylacetonate (Fe(acac) 3 ) (complex 2 ) and chloro(protoporphyrinato)iron(III) (hemin) (complex 3 Fig

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NFE2L2 mRNA levels stay constant after exposure to DEM, indicating that Nrf2 is not positively regulating itself by expression of NFE2L2 mRNA

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