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Secondly, the direct mechanism of action and targets against YAP signaling were not further validated in vivo experiments

In vitro experiments and neonatal animal models of HI suggested a very complex network of cell death cascades, highlighting a continuum from apoptosis to necrosis 33,34

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S-sulfocysteine can function as an NMDAR agonist to aggravate sulfite neurotoxicity in the brain, promote depolarization of presynaptic membrane sites, and induce a large Ca 2+ influx [75]

Chem Res Toxicol 2(1):4145 Rashed MS, Myers TG, Nelson SD (1990) Hepatic protein arylation, glutathione depletion, and metabolite profiles of acetaminophen and a non-hepatotoxic regioisomer, 3-hydroxyacetanilide, in the mouse