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Specifically, the pathologic effects of anti-GAD65Abs on GABA synthesis and release were specific to anti-GAD65Ab b78representing SPS- and CA-associated anti-GAD65Aband could not be reproduced by monoclonal anti-GAD65Ab b96.11, representing an epitope specificity associated with type 1 diabetes mellitus [80, 81, 82]

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Pharmacological approach to the mechanisms of transcranial DC-stimulation-induced after-effects of human motor cortex excitability