Its primary mechanism is the upregulation of telomerase, the enzyme responsible for maintaining and extending telomeres, the protective end-caps on DNA strands
Enhanced angiogenesis could speed this compensatory process
The oxidative damage to these targets can lead to altered protein structure, localization, activity and interaction networks, epigenetic modification to DNA chromatin structure, disrupted membrane lipid integrity, mitochondria dysfunction, and even apoptosis (Avery, 2011
Methionine, in the form of S-adenosylmethionine (SAM), is required for many methylation reactions, including DNA methylation
This allows the glutathione liposomal formulation to pass the digestive system and reach the bloodstream effectively without breaking down