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The purpose of this study was to address these interpretational challenges through meta-analytic techniques in the service of clarifying the impact of chronic smoking on gray matter integrity and more fully contextualizing such structural alterations

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In non-small-cell lung cancer (NSCLC) or breast cancer cells, nicotine treatment stimulated Src and Raf signalling, resulting in Rb phosphorylation and subsequent cell cycle progression (Nakayama et al, 2002

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