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(2) We have already found that Nrf2 is the key mediator in the protective effect of AZI on airway epithelial barrier, but how AZI activates Nrf2 is still unknown, which is our next research objective

This may also suggest that exposure to e-cigarette vapor chemicals may have differential effects under homeostatic conditions (increased MAPK activation) versus inflammatory settings (ulcerative colitis)(39)

Older studies involving high-dose estrogen OCs showed a significant interaction with smoking, increasing the risk for stroke and SAH among women who both smoked and used OCs