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These inbred mice often develop spontaneous benign tumors in the lung (adenomas) that may, on occasion, progress to cancerous lesions (adenocarcinomas)

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Use of the PARP-1 inhibitor, BMN673, reduced AIF and EndoG translocation after smoke exposure, confirming the role of PARP-1 activation in smoke induced activation of the parthanatos pathway in primary HBE cells