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Suggested causes and mechanisms include immune-mediated responses, altered drug metabolism associated with glutathione deficiency or slow acetylator phenotype, co-existing infections with cytomegalovirus (CMV) or Epstein-Barr virus (EBV), or cytokines involvement

2, 2011, pp

The data demonstrate that acTrxR1 K141 and acTrxR1 K200 are significantly more resistant to oxidation than the un-modified enzyme