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However, despite these similarities, the functions between IGF-1R and IR differ considerably

About this article Cite this article Wei, Y., Qian, H., Zhang, X

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This previous study also revealed that this phosphorylation is driven by E2 and is not inhibited by estrogen receptor antagonists (104), suggesting a potential alteration in AKT signaling in endometriosis, making it more responsive to estrogen or resistant to conventional antagonism

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