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By disrupting the interaction between FOXO4 and p53, FOXO4-DRI induces apoptosis (programmed cell death) in senescent cells, reducing their harmful effects

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Once the S-A-GSH molecule is safely inside the target cell, specialized enzymes called intracellular thioesterases recognize the molecule and cleave (remove) the acetyl group

Significant acceleration in wound repair was evident on Days 7 and 11 ( p 2log 10 within 12 hours [168]

Adding exogenous glutathione to a drug that already modulates oxidative stress raises a conceptual question: could the combined antioxidant load suppress reactive oxygen species (ROS) to a degree that interferes with beneficial ROS signaling