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In summary, GSH enables antibiotic inactivation by serving as a substrate for detoxifying enzymes, such as FosA, and by modulating the expression or stability of antibiotic-degrading enzymes, including the downregulation of NDM-1

Thus, an illustration is the above-mentioned neuroleptic-domperidone-amphetamine occlusion/occlusion-like syndrome [54], in particular (i.e., haloperidol, fluphenazine, clozapine, risperidone, olanzapine, quetiapine, aripiprazole, domperidone, amphetamine, and combined amphetamine and haloperidol all produced similar syndrome)

The study also reported reproductive and developmental toxicity in ICR mice exposed to PE MPs chronically [15]

Maes, M., Almulla, A

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