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[2] [9] However, other research suggests that the proportion of people with low or marginal levels of vitamin B 12 is up to 40% in the Western world

The hypothesis that B12 could modulate CCL11 expression during persistent VCD via methyl-dependent epigenetic mechanisms was confirmed by the hypermethylation of DML a and c in leukocytes from cultures of impaired individuals incubated with the vitamin