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The pharmacokinetic barrier has recently been overcome with the synthesis of the orally active, blood-brain barrierpermeable analog N-hexanoic-tyrosine-isoleucine-(6) aminohexanoic amide (dihexa)

But these methods have shortcomings: Working with radioactivity is cumbersome and hazardous, and antibodies aren't available for some phosphorylation sites

GHK-Cu is generally stable but can potentially be deactivated or cause irritation when combined with certain strong acids (like high concentrations of AHAs or BHAs, particularly Ascorbic Acid/Vitamin C in low pH formulations) and some direct acids (like L-Ascorbic Acid)

Keener et al., 2021