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However, several models of TBI in rodents demonstrate a reduction in amyloid beta plaques following TBI (60, 61), and one study showed that mice overexpressing amyloid precursor protein had a rise in unaggregated A in the hippocampus with extensive hippocampal neuronal death, thereby suggesting that the plaques may be protective against unaggregated (A) toxicity unclear (62), though it remains

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