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As illustrated in Figure 3, pro-inflammatory cytokines, including TNF-, IL-1, and IL-6, promote OA progression by activating signaling intermediates such as p38 and ERK1/2, leading to upregulation of matrix metalloproteinases (MMPs) and degradation of cartilage structure

Assessment of the State of the Natural Antioxidant Barrier of a Body in Patients Complaining about the Presence of Tinnitus

Future Research directions In-depth Analysis of the Mechanism of Action Adipose tissue microenvironment: Exploring the effects of AOD 9604 on inflammatory factors and angiogenesis in adipose tissue

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