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62 FAK-Paxillin Modulation: It modulates focal adhesion kinase (FAK) and paxillin complexes, which are essential for fibroblast migration and survival at wound sites

Long-term deficiency might lead to irreversible damage to the brain and nervous system

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Over five decades of peer-reviewed research have established GHK-Cu as a multi-pathway regenerative compound with documented activity in collagen synthesis, wound closure, anti-inflammatory signaling, antioxidant defense, hair follicle biology, and broad gene expression modulation