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The translational promise of this approach lies in the development of next-generation anti-inflammatory agents with greater central nervous system specificity and improved safety profiles

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[4] Complementary work has reported that Semax and its Pro-Gly-Pro fragment together activate transcription of neurotrophins and their receptor genes after ischemia, reinforcing the picture of a peptide that acts partly by nudging endogenous neurotrophic and inflammatory programs

Via our first nonoxidative reaction, phosphopentose isomerase catalyzes a rearrangement (an isomerization) of ribulose 5-phosphate to ribose 5-phosphate