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established a chemical cocktail of CHIR, A8301, and SAG (a Hedgehog signaling activator) combined with EGF, HGF and BMP4, which significantly promoted the expansion and stemness maintenance of HPCs (Pan et al., 2019)

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Conversely, following hypoxia or loss of mitochondrial membrane potential, the inactivation or reduction of tyrosine kinase activity through the activation of PGAM5 will contribute to the downregulation of the phosphorylated FUNDC1 level

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