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ERK1/2 activation upregulates collagen gene expression and promotes cell proliferation critical for fibroblast proliferation, while p38 MAPK inhibition reduces inflammatory response

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Such a tool may incorporate key risk factors delineated in the present review, including arthritis disease activity, comorbidities associated with an elevated risk of ED, metabolic indicators linked to endocrine-metabolic dysregulation, and demographic characteristics (e.g., age, body mass index) consistently correlated with ED in arthritis cohorts