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This study was approved by the local ethics committee of the Hannover Medical School (Vote 4253) and was conducted according to the Declaration of Helsinki Principles

Endoplasmic reticulum (ER) stress and hypoxia response pathways interact to potentiate hypoxia-inducible factor 1 (HIF-1) transcriptional activity on targets like vascular endothelial growth factor (VEGF)

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By combining BPC-157 (Body Protection Compound) and TB-500 (Thymosin Beta-4), this protocol targets two distinct but complementary pathways of healing: angiogenesis (blood vessel formation) and cellular migration