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[2] Broader reviews position the peptide as producing an angiogenic, healing-promoting effect across gastrointestinal and extra-gastrointestinal tissues, tying its action to the nitric-oxide system and to standard angiogenic growth factors

The Arg3 substitution and N-terminal 13-residue extension in IGF-1 LR3 reduce IGFBP affinity by approximately 1,000-fold, preventing this sequestration