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Interestingly, we observed that the cecal microbiome of previously-obese animals returned to the lean state through NNMTi treatment and LD switch was distinct from the cecal microbiome of continuously lean control animals

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Amyloid and tau pathology of familial Alzheimers disease APP/PS1 mouse model in a senescence phenotype background (SAMP8)

Again, took me a few months to figure that portion out how to make that part

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