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Findings: Preclinical models demonstrated enhanced anabolic signaling and support for tissue recovery pathways

The development of rheumatoid arthritis is based on a genetic predisposition, such as the HLA-DR4 alleles, that create antibodies against citrullinated proteins, and on environmental triggers such as infection or smoking [8]

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Laboratory studies have also shown that it can reduce the body's response to acute stress, as it significantly affects the levels of substance P, beta-endorphin and cortisol in the hypothalamus and blood plasma

In particular, GSTP1-1 is able to protect the tumor cells from apoptosis signals by inhibiting c-Jun N-terminal kinase (JNK)a member of the MAPK pathwaynon-catalytically, by direct proteinprotein association 25,26