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The 177191 fragment was identified as the lipolytic active site, and a tyrosine residue was added at the N-terminus to enhance stability

Deficiency can also be caused by poor absorption

Promoting new blood vessel growth (angiogenesis): BPC-157 appears to stimulate the growth of new blood vessels through pathways involving VEGFR2 and nitric oxide, delivering more oxygen and nutrients to the injury site

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