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The target genes of p53 involve the canonical ferroptosis pathway of GSH metabolism (e.g., SLC7A11), iron metabolism (TfR1 and SLC25A28), lipid metabolism, ROS generation, several oxidation/reduction related proteins, and the non-canonical ferroptosis pathway (66, 67)

An ensemble of the ten lowest energy structural models is shown in Fig

These supersulfides, regardless of their forms, can be generated via enzymatic or chemical processes

Thus, we speculate that the basal levels of tissue-resident macrophages in WT and Klf9 -null endometria may contribute to the distinct lesion parameters associated with the same HFD environment

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