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Endocrinology 148 (10), 46584666

It drives the transition from proliferation to differentiation and increases the number and length of multinucleated myotubes in a dosedependent manner.The mechanism is mainly related to NNMT inhibition, restored NAD levels, and activation of the SIRT1PGC1 pathway

M., Sun, J., Sharma, L., Libinaka, R., Jiang, W

The hormone promotes gluconeogenesis (glucose production) in the liver and reduces glucose uptake in peripheral tissues, creating an insulin-resistant state

Whilst mTORC1 activates S6 kinase, which phosphorylates serine residues on IRS which in turn uncouples IRS from the receptor and its substrates, mTORC2 can reactivate this signalling