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Moreover, overexpression of cGAS in RA-FLSs enhanced both the proliferation of these cells and the expression of pro-inflammatory factors ( Sting1 +/ mice, which have a reduced expression of phosphorylated TBK1, showed a decreased severity of arthritis and improved histological changes compared to control mice ( Figure 2 )

Antibiotic drug use and the risk of Stevens-Johnson Syndrome and Toxic Epidermal Necrolysis: a population-based case-control study

In conclusion, mitochondria function as central hubs for energy metabolism and signaling, interacting with cellular organelles to meet the energy requirements and antioxidant defense needs during follicular development