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This review aims to highlight the central role of OS in the pathophysiology of ALS, specifically focusing on the KEAP1-NRF2 system and its alteration in ALS, while discussing the potential applications of NRF2 activators in ALS therapy

Psychopharmacology (Berl) 2006;186(4):487503

Insufficient concentrations of glutathione may increase the accumulation of ROS, including that of the highly reactive hydroxyl radical, which plays a key role in membrane lipid peroxidation and cellular structure damage [64]

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