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Results The clinical appearance showed that severe, moderate, and mild degrees accounted for 42%, 45%, and 13%, respectively

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The latest research indicates that I3A not only sustains lung epithelial barrier function through AhR receptor activation and IL-22 secretion stimulation but also inhibits HDAC5/6, resulting in the inactivation of the NF-B/NLRP3 signaling pathway and the subsequent reduction of inflammatory factor expression (66)

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These two drugs were not only able to enrich GSH contents and GPX activity and defend the heart against oxidative stress but also prevented DOX or I/R-induced myocardial injury [214,215,216,217]