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Gores, G

An interaction between parkin and its substrate -synuclein is a crucial regulatory component of dopaminergic neuron degeneration, loss-of-function mutations in PARK2 usually cause early-onset autosomal recessive PD (Kitada et al., 1998), whereas disease-associated mutations in -synuclein are usually associated with autosomal dominant PD (Polymeropoulos et al., 1997)

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While this approach is established for other nutrients, no robust human trials yet demonstrate that liposomal BPC-157 produces superior clinical outcomes compared with non-liposomal forms