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[8] The scientists suggest that GHRH-A [appeared to have] improved cardiac diastolic hemodynamics, including end-diastolic pressure, end-diastolic pressurevolume ratio, and stroke work, compared to placebo. Some laboratory studies reported a decrease in infarct size and attenuation of cardiac hypertrophy in mice

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Furthermore, our studies were limited to male mice if these findings apply to females, which have been demonstrated to be inherently resistant to ferroptosis upon IRI [6], should be addressed in future

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