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Their translational attractiveness is nevertheless tempered by several practical issues

The role of Nrf2 in liver fibrosis has been clarified by Lu, who showed that GCL subunits and GS are regulated byNrf2, which binds AREs present in their promoters [2] (Figure 6)

In addition, SA has been shown to stabilize auxin repressor protein AUX/IAA by inhibiting the expression of TIR1/AFB transport inhibitor resistant1)/(auxin signaling F-BOX), thereby inhibiting auxin response and enhancing plant resistance [81]

TM6SF2: A Novel Genetic Player in Nonalcoholic Fatty Liver and Cardiovascular Disease