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found that ferroptosis in ectopic endometrial stromal cells in patients with ovarian endometriosis could activate the p38 mitogen-activated protein kinase (p38 MAPK)/signal transducer and activator of transcription (STAT) 6 signalling pathway, thereby promoting local upregulation of vascular endothelial growth factor A (VEGFA) and interleukin-8 (IL-8) in ectopic lesions [15]

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This pharmacokinetic difference explains why IGF-1 LR3 is preferred in experimental research to study direct effects of IGF-1R signaling without confounding biases linked to complex IGFBP biology

& Kadlubar, F