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Fibrosis in Non-Alcoholic Steatohepatitis In addition to exacerbating hepatocyte injury and apoptosis, activation of Kupffer cells and expansion of the inflammatory niche stimulates differentiation of liver-resident HSCs into pro-fibrogenic myofibroblasts (192) ( Figure 1B )

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Loss of presenilin function causes impairments of memory and synaptic plasticity followed by age-dependent neurodegeneration