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Another significant concern is that excessive modulation of the Th17/Treg balance carries potential risks, as it may damage the hosts immune defense against pathogens or destroy self-tolerance

[1] [2] This triple-receptor mechanism distinguishes it from currently approved agents such as semaglutide (a GLP-1 agonist) and tirzepatide (a dual GIP/GLP-1 agonist)

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Patient selection should prioritize individuals with conditions where established therapies have proven inadequate or are contraindicated

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