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The peptide achieves these complex neurological stabilizations largely through intricate, bidirectional interactions with the nitric oxide (NO) system, dynamically responding to both NO-system blockade (via L-NAME administration) and NO-system over-stimulation (via L-arginine administration) to restore neurological homeostasis

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VEGF binds VEGFR2 on endothelial cells triggering proliferation, migration, and tube formation creating new blood vessels