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The pathophysiology of doxorubicin and ischemia/reperfusion-induced myocardial injury in mice may be primarily associated with increased mitochondrial iron levels and lipid peroxidation, suggesting that mitochondrial oxidative damage is a principal mechanism underlying ferroptosis in this context [107]

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The synthetic version, semaglutide, has been modified with a fatty acid side chain that allows it to bind to albumin in the blood