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Given the coupling between copper and energy status (e.g copper is a cofactor at Complex IV of the ETC) and how intensely metabolically active neurons are, it seems like short-term IP GHK-cu in the brain is acting analogously to the copper ionophore elesclomol in Tregs

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Naughton et al., 2020), and also takes into account the possible downstream mechanisms such as cuproenzyme dysfunction which we believe is highly important to include for a more complete picture of AD