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Dehghani F, Sezavar Seyedi Jandaghi SH, Janani L, Sarebanhassanabadi M, Emamat H, Vafa M

A can promote the release of Glu from vesicles into the synaptic cleft, leading to the activation of extrasynaptic N-methyl-D-aspartate receptors (NMDARs)

Research areas include tissue repair mechanisms, angiogenesis, cytoprotection, and interaction with the nitric oxide system

Semax provides direct neurotrophic stimulation and BDNF upregulation, while Selank adds anxiolytic effects through GABAergic modulation and additional BDNF support through serotonergic pathways

Astroglial and microglial contributions to iron metabolism disturbance in Parkinson's disease